Bacterial mediators of endometriosis

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AI-generated summary by claude@2026-06+body, 2026-06-13

This research highlight discusses how bacterial mediators may explain the pathogenic activation of stromal fibroblasts, which are implicated in endometriosis.

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This research highlight discusses endometriosis as a common, painful condition caused by endometrium-like tissue growing outside the uterus, focusing on stromal fibroblasts as potential mediators whose pathogenic activation has been difficult to explain. It frames the central topic as bacterial mediators of endometriosis, implying an evidence-based link between microbial factors and how fibroblasts may become activated in disease. The limitation is that, as presented here, the piece functions as a highlight rather than a full mechanistic or experimental study with detailed methods and results. This paper is centrally about endometriosis — it specifically addresses bacterial mediators that may drive pathogenic activation of stromal fibroblasts in endometriosis.

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- RESEARCH HIGHLIGHT Bacterial mediators of endometriosis Endometriosis is a painful and common condition caused by endometrium-like tissue growing outside the uterus. Stromal fibroblasts (among other things) have been implicated as potential mediators, but an explanation for their pathogenic activation has been elusive. Access options Access Nature and 54 other Nature Portfolio journals Get Nature+, our best-value online-access subscription 27,99 € / 30 days cancel any time Subscribe to this journal Receive 12 print issues and online access 251,40 € per year only 20,95 € per issue Rent or buy this article Prices vary by article type from$1.95 to$39.95 Prices may be subject to local taxes which are calculated during checkout doi: https://doi.org/10.1038/d41591-023-00057-z

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endometriosis

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europepmc
last seen: 2026-06-21T06:12:49.409960+00:00
openalex
last seen: 2026-06-10T17:14:06.276822+00:00
pubmed
last seen: 2026-06-21T06:11:43.528101+00:00
unpaywall
last seen: 2026-06-13T06:42:57.164913+00:00
License: CC0 · commercial use OK