INFRINGEMENT OF ANGIOGENESIS AND SCLEROSIS REGULATION PROCESSES IN HYPERPLASTIC ENDOMETRIUM
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Increased VEGF, TGFβ1, fibronectin, and lysyl oxidase in hyperplastic endometrium indicate cellular hypoxia and disrupted cell interactions, suggesting a negative prognostic factor.
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Abstract
Pathomorphological and immunohistochemical studies of endometrium were performed. The higher proliferative activity of the VEGF and the higher production of transformation growth factor β1 (TGFβ1), fibronectin and liziloxydaza in patiens with atypical hyperplasia of endometrium and chronic endometritis testifies the presence of cellular hypoxia, disturbance of intercellular and extracellular interaction and can be considered as the negative prognostic factor.
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- last seen: 2026-06-10T17:14:06.276822+00:00
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