Biology of Normal Aging Endometrium

R Trévoux, J. De Brux, C. Bergeron
In: Clinical Perspectives in Obstetrics and Gynecology · 1994 · pp. 246–253 · doi:10.1007/978-1-4612-4330-4_24 · W176698884
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⚙ AI-generated summary by claude@2026-06+body, 2026-06-10 ⓘ

The postmenopausal endometrium can regenerate, exhibiting individual variations in evolution even within the same woman, with spontaneous hyperplasia evolving similarly to normal endometrium.

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⚙ AI-generated deep summary by claude@2026-06, 2026-06-10 ⓘ

This chapter reviews the biology of the normal postmenopausal endometrium, focusing on how an atrophic endometrium can retain regenerative properties through resumed activity of quiescent ovarian follicles or through estrogen replacement, while noting that nonhormonal “estrogen-like” therapies can also produce paradoxical stimulation; it also describes that endometrial behavior may appear autonomous when out of phase with biological data. It reports a 13-year longitudinal study of 863 women not on hormone replacement, using annual endometrial biopsies correlated in 283 cases with gonadal steroid hormones and gonadotropins collected 6 years before and 7 years after amenorrhea, showing substantial inter- and intra-individual variation in endometrial evolution. The authors further state that untreated simple hyperplasias without cytologic atypia spontaneously evolve identically to normal endometrium. This paper does not explicitly discuss endometriosis or adenomyosis; it was included in the corpus via a keyword match in the upstream search index.

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