MicroRNAs and breast cancer malignancy: an overview of miRNA-regulated cancer processes leading to metastasis.

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This review outlines recent findings on how specific microRNAs regulate gene expression to drive breast cancer malignancy, focusing on their roles in metastasis, EMT/MET, migration, invasion, and angiogenesis.

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This review examines the role of microRNAs in regulating gene expression and driving malignancy processes in breast cancer, with a specific focus on metastatic mechanisms. The authors analyze how specific miRNAs influence epithelial-to-mesenchymal transition, cell migration, invasion, and angiogenesis to promote cancer aggressivity. While the paper outlines these molecular pathways, it is a narrative review that summarizes existing literature rather than presenting new experimental data. Relevance to endometriosis: listed as one indication for GnRH antagonists, though the paper's main focus is uterine fibroids.

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Abstract

Cancer statistics show significant diagnosis numbers amongst men and women worldwide, where breast cancer is by far the most frequently diagnosed cancer in women. Multiple mechanisms and molecules have been shown to occupy major roles in cancer progression and aggressivity. Recently, small non-coding RNA molecules, called micro-RNAs, have become the subject of interest in many molecular pathways in relation to breast cancer, amongst many other pathologies. MiRNAs are capable of regulating gene expression in a sequence-specific manner and regulate diverse expression patterns which are dependent on the cell's state and identity. Studies have brought forward specific miRNAs that have the innate ability to govern unique gene expression profiles regulating cancer cell aggressivity. This review will outline recent findings of characterized miRNAs in relation to their molecular targets leading to cancer malignancy and progression. More specifically, we will focus on miRNAs associated with breast cancer metastatic processes including epithelial to mesenchymal and mesenchymal to epithelial transitioning (EMT/MET transition), migration, invasion and angiogenesis.
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Abstract

Cancer statistics show significant diagnosis numbers amongst men and women worldwide, where breast cancer is by far the most frequently diagnosed cancer in women. Multiple mechanisms and molecules have been shown to occupy major roles in cancer progression and aggressivity. Recently, small non-coding RNA molecules, called micro-RNAs, have become the subject of interest in many molecular pathways in relation to breast cancer, amongst many other pathologies. MiRNAs are capable of regulating gene expression in a sequence-specific manner and regulate diverse expression patterns which are dependent on the cell’s state and identity. Studies have brought forward specific miRNAs that have the innate ability to govern unique gene expression profiles regulating cancer cell aggressivity. This review will outline recent findings of characterized miRNAs in relation to their molecular targets leading to cancer malignancy and progression. More specifically, we will focus on miRNAs associated with breast cancer metastatic processes including epithelial to mesenchymal and mesenchymal to epithelial transitioning (EMT/MET transition), migration, invasion and angiogenesis.

Keywords

miRNA, breast cancer, metastasis, migration, invasion, EMT, MET, angiogenesis Full Text The Full Text of this article is available as a PDF (181.3 KB).

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