High mobility group box 1 promotes endometriosis under hypoxia by regulating inflammation and autophagy in vitro and in vivo

Jingying Huang, Xuan Chen, Jiangrui Liu
International immunopharmacology · 2023 · vol. 127 , pp. 111397 · doi:10.1016/j.intimp.2023.111397 · PMID:38134596 · W4390086504
article OA: closed CC0 ⤵ 4 in-corpus citations
Limited metadata. Only one source feed has indexed this record so far — no abstract, full text, or open-access copy is available through Endo Lab. The publisher's page (linked below) is the canonical location for the actual content. If you have institutional access, use "Find at my library".
View at publisher → View on OpenAlex View on PubMed
⚙ AI-generated summary by claude@2026-06, 2026-06-08 ⓘ

High mobility group box 1 (HMGB1) promotes endometriosis by regulating inflammation and autophagy in both cell cultures and animal models under hypoxic conditions.

One-sentence paraphrase of the abstract; not a substitute for reading it. No clinical advice. How this works

My notes (saved in your browser only)

Condition tags

endometriosis

MeSH descriptors

Endometriosis Endometriosis Endometriosis HMGB1 Protein HMGB1 Protein HMGB1 Protein HMGB1 Protein HMGB1 Protein Animals Animals Animals Autophagy Autophagy Autophagy Cytokines Cytokines Cytokines Cytokines Female Female

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (42)

Cited by (4)

Source provenance

europepmc
last seen: 2026-06-11T06:19:48.454388+00:00
openalex
last seen: 2026-06-10T17:14:06.276822+00:00
pubmed
last seen: 2026-05-25T00:33:13.492766+00:00
unpaywall
last seen: 2026-06-02T02:00:03.124865+00:00
License: CC0 · commercial use OK