Wang, Yi-Yi

No ORCID on file · 4 papers in corpus · active 2021

Study types

  • other 4

Condition tags

  • adenomyosis 4
other 2021
Additional file 1 of Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
·doi:10.6084/m9.figshare.14600554.v1

Additional file 1: Supplementary Figure S1. Morphological features of adenomyotic eutopic and ectopic endometrial stromal cells. A.and B. The primary culture of ADS_Eu_ESC and ADS_Ec_ESC cells to the fourth day, respectively. C. The third g…

other 2021
Additional file 1 of Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
·doi:10.6084/m9.figshare.14600554

Additional file 1: Supplementary Figure S1. Morphological features of adenomyotic eutopic and ectopic endometrial stromal cells. A.and B. The primary culture of ADS_Eu_ESC and ADS_Ec_ESC cells to the fourth day, respectively. C. The third g…

other 2021
Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
·doi:10.6084/m9.figshare.c.5425468.v1

Abstract Adenomyosis (ADS) is an estrogen-dependent gynecological disease with unspecified etiopathogenesis. Local hyperestrogenism may serve a key role in contributing to the origin of ADS. Talin1 is mostly identified to be overexpressed a…

other 2021
Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
·doi:10.6084/m9.figshare.c.5425468

Abstract Adenomyosis (ADS) is an estrogen-dependent gynecological disease with unspecified etiopathogenesis. Local hyperestrogenism may serve a key role in contributing to the origin of ADS. Talin1 is mostly identified to be overexpressed a…