Russell R. Broaddus

ORCID: 0000-0002-8692-273X · 18 papers in corpus · active 2003-2023

Study types

  • preprint 14
  • article 4

Condition tags

  • adenomyosis 2
preprint 2023
Supplementary Figure 1 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443752

PDF file - 387K, Three-lineage differentiation of O-ASC.

preprint 2023
Supplementary Figure 1 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443752.v1

PDF file - 387K, Three-lineage differentiation of O-ASC.

preprint 2023
Supplementary Figure 2 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443749

PDF file - 678K, O-ASC do not co-localize with CD31 positive endothelial cells.

preprint 2023
Supplementary Figure 2 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443749.v1

PDF file - 678K, O-ASC do not co-localize with CD31 positive endothelial cells.

preprint 2023
Supplementary Figure 3 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443746

PDF file - 202K, Quantification of histological and immunofluorescence tissue analysis.

preprint 2023
Supplementary Figure 3 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443746.v1

PDF file - 202K, Quantification of histological and immunofluorescence tissue analysis.

preprint 2023
Supplementary Figure 4 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443743.v1

PDF file - 139K, Endometrial Hec1a cancer cell proliferation in co-culture with OSC, MSC, ASC and WI38 fibroblasts.

preprint 2023
Supplementary Figure 4 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443743

PDF file - 139K, Endometrial Hec1a cancer cell proliferation in co-culture with OSC, MSC, ASC and WI38 fibroblasts.

preprint 2023
Supplementary Figure 6 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443737

PDF file - 99K, Migration of stromal cells in response to Hec1a conditioned media.

preprint 2023
Supplementary Figure 6 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443737.v1

PDF file - 99K, Migration of stromal cells in response to Hec1a conditioned media.

preprint 2023
Supplementary Figure 7 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443734

PDF file - 281K, Hec1a secrete IL8 and CXCL1.

preprint 2023
Supplementary Figure 7 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443734.v1

PDF file - 281K, Hec1a secrete IL8 and CXCL1.

preprint 2023
Supplementary Figure 8 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443731

PDF file - 1.0MB, Expression of IL8 / CXCL1 receptors in mesenchymal cells.

preprint 2023
Supplementary Figure 8 from Omental Adipose Tissue–Derived Stromal Cells Promote Vascularization and Growth of Endometrial Tumors
·doi:10.1158/1078-0432.22443731.v1

PDF file - 1.0MB, Expression of IL8 / CXCL1 receptors in mesenchymal cells.

article 2018
MIG-6 suppresses endometrial epithelial cell proliferation by inhibiting phospho-AKT
·doi:10.1186/s12885-018-4502-7

BACKGROUND: Aberrant hyperactivation of epithelial proliferation, AKT signaling, and association with unopposed estrogen (E2) exposure is the most common endometrial cancer dysfunction. In the normal uterus, progesterone (P4) inhibits proli…

article 2013
β ‐Catenin activation contributes to the pathogenesis of adenomyosis through epithelial–mesenchymal transition
The Journal of Pathology ·doi:10.1002/path.4224

Adenomyosis is defined by the presence of endometrial glands and stroma within the myometrium. Despite its frequent occurrence, the precise aetiology and physiopathology of adenomyosis is still unknown. WNT/β-catenin signalling molecules ar…

article 2010
Abstract B23: Dominant stablized ß-catenin induces adenomyosis formation and ablation of Mig-6 accelerates progress of adenomyosis formation
·doi:10.1158/1078-0432.tcmusa10-b23

Abstract Adenomyosis is a common gynecological disorder defined by the presence of endometrial glands and stroma within the myometrium. Despite its frequent occurrence, the precise etiology of adenomyosis is still unknown, although it has o…

article 2003
Inhibition of proliferation and estrogen receptor signaling by peroxisome proliferator-activated receptor gamma ligands in uterine leiomyoma.

Peroxisome proliferator-activated receptor (PPAR) gamma is an important signaling molecule in cells of mesenchymal origin, inducing differentiation and regulating cell proliferation in several cell types such as vascular smooth muscle cells…