Amy E. Birsner

ORCID: 0000-0002-4308-0966 · 3 papers in corpus
article 2013
Vascular endothelial growth factor C is increased in endometrium and promotes endothelial functions, vascular permeability and angiogenesis and growth of endometriosis
Angiogenesis ·doi:10.1007/s10456-013-9333-1

Endometriosis is an angiogenesis-dependent disease. Many studies demonstrated inhibition of angiogenesis leads to inhibition of endometriotic growth, however underlying mechanism is still not fully understood. Our previous study suggested v…

other 2011
Green tea epigallocatechin-3-gallate inhibits angiogenesis and suppresses vascular endothelial growth factor C/vascular endothelial growth factor receptor 2 expression and signaling in experimental endometriosis in vivo
Fertility and sterility ·doi:10.1016/j.fertnstert.2011.07.008

OBJECTIVE: To investigate the antiangiogenesis mechanism of epigallocatechin-3-gallate (EGCG) in an endometriosis model in vivo. DESIGN: Animal studies. SETTING: University laboratory. ANIMAL(S): Human endometrium from women with endomet…

other 2008
Differential suppression of vascular permeability and corneal angiogenesis by nonsteroidal anti-inflammatory drugs
Investigative ophthalmology & visual science ·doi:10.1167/iovs.07-1527

PURPOSE: Angiogenesis, the formation of new capillary blood vessels, is an essential biological process under physiological conditions, including embryonic development, reproduction, and wound repair. Under pathologic conditions, this proce…