Li Wang (15202)

No ORCID on file · 14 papers in corpus · active 2022-2026

Study types

  • dataset 9
  • other 4
  • article 1

Condition tags

  • endometriosis 8
other 2026
·doi:10.1371/journal.pone.0350751.g007

A. Enrichment plots for the top 4 biological functions enriched in the endometriosis group of the combined dataset. B-E. GSEA enrichment plots demonstrating the significant impact of endometriosis on the following pathways: IL1 and Megakary…

other 2026
·doi:10.1371/journal.pone.0350751.s013

A. Box plots illustrating the differential enrichment of hallmark pathways between the high and low pyroptosis expression groups. B. Heatmap depicting the enrichment scores of hallmark pathways in the two groups. (PNG)

article 2026
·doi:10.1371/journal.pone.0350751.s004

To investigate the differential enrichment of hallmark pathways between the high and low pyroptosis expression groups in endometriosis, GSVA was performed using the expression data of all genes in the combined dataset. The results of the GS…

other 2026
·doi:10.1371/journal.pone.0350751.g011

A. Consensus clustering matrix for endometriosis samples, demonstrating the stability of the identified subtypes. B. Consistent Cumulative Distribution Function (CDF) plot for the consensus clustering analysis, indicating the optimal number…

other 2026
·doi:10.1371/journal.pone.0350751.g010

A. Box plots comparing the abundance of immune cell subtypes identified by CIBERSORT between endometriosis and normal subgroups. B. Box plots comparing the ESTIMATE scores (Estimate Score, Immune Score, and Stromal Score) between endometrio…

dataset 2026
·doi:10.1371/journal.pone.0350751.t001

Background Endometriosis (EMs) is a chronic inflammatory disease characterized by ectopic endometrial growth. This study aimed to identify and analyze potential signatures of pyroptosis-related genes in EMs. Methods We conducted a comprehe…

dataset 2022
·doi:10.3389/fendo.2022.858176.s001

BackgroundEndometriosis (EMS), an endocrine-related inflammatory disease, is characterized by estrogen and progesterone imbalance in ectopic lesions. However, its pathogenic mechanism has not been fully elucidated. While SCM-198 is the synt…

dataset 2022
·doi:10.3389/fendo.2022.858176.s002

BackgroundEndometriosis (EMS), an endocrine-related inflammatory disease, is characterized by estrogen and progesterone imbalance in ectopic lesions. However, its pathogenic mechanism has not been fully elucidated. While SCM-198 is the synt…

dataset 2022
·doi:10.3389/fendo.2022.858176.s003

BackgroundEndometriosis (EMS), an endocrine-related inflammatory disease, is characterized by estrogen and progesterone imbalance in ectopic lesions. However, its pathogenic mechanism has not been fully elucidated. While SCM-198 is the synt…

dataset 2022
·doi:10.3389/fendo.2022.858176.s004

BackgroundEndometriosis (EMS), an endocrine-related inflammatory disease, is characterized by estrogen and progesterone imbalance in ectopic lesions. However, its pathogenic mechanism has not been fully elucidated. While SCM-198 is the synt…

dataset 2022
·doi:10.3389/fendo.2022.858176.s005

BackgroundEndometriosis (EMS), an endocrine-related inflammatory disease, is characterized by estrogen and progesterone imbalance in ectopic lesions. However, its pathogenic mechanism has not been fully elucidated. While SCM-198 is the synt…

dataset 2022
·doi:10.3389/fendo.2022.858176.s006

BackgroundEndometriosis (EMS), an endocrine-related inflammatory disease, is characterized by estrogen and progesterone imbalance in ectopic lesions. However, its pathogenic mechanism has not been fully elucidated. While SCM-198 is the synt…

dataset 2022
·doi:10.3389/fendo.2022.858176.s007

BackgroundEndometriosis (EMS), an endocrine-related inflammatory disease, is characterized by estrogen and progesterone imbalance in ectopic lesions. However, its pathogenic mechanism has not been fully elucidated. While SCM-198 is the synt…

dataset 2022
·doi:10.3389/fendo.2022.858176.s008

BackgroundEndometriosis (EMS), an endocrine-related inflammatory disease, is characterized by estrogen and progesterone imbalance in ectopic lesions. However, its pathogenic mechanism has not been fully elucidated. While SCM-198 is the synt…