Warren B. Nothnick

2001

Eutopic endometrium from women with endometriosis express a novel 41 kDa protease and elevated stromelysin activity: implications into the pathophysiology of endometriosis.

·doi:10.1016/s0015-0282(01)02152-5

review 2001

Treating endometriosis as an autoimmune disease

Fertility and sterility ·doi:10.1016/s0015-0282(01)01878-7

OBJECTIVE: To review the literature on the role of autoimmunity in the etiology of endometriosis, compare the similarities in the pathophysiologies between endometriosis and autoimmune diseases, and discuss the use of immunomodulators curre…

other 1998

Gonadotropin-releasing hormone agonist (GnRH-a) therapy alters activity of plasminogen activators, matrix metalloproteinases, and their inhibitors in rat models for adhesion formation and endometriosis: potential GnRH-a-regulated mechanisms reducing adhesion formation

Fertility and sterility ·doi:10.1016/s0015-0282(98)00032-6

OBJECTIVE: To evaluate the effects of a gonadotropin-releasing hormone agonist (GnRH-a) on plasminogen activator (PA), matrix metalloproteinase (MMP), plasminogen activator inhibitor (PAI) and matrix metalloproteinase inhibitor (MMPI) activ…

1998

Novel implications in the development of endometriosis: Biphasic effect of immunostimulation on peritoneal tissue expression of Tissue Inhibitor Of Metalloproteinase-1 (TIMP-1)

·doi:10.1016/s1071-5576(97)86278-8

lab-animal 1998

Novel implications in the development of endometriosis: biphasic effect of macrophage activation on peritoneal tissue expression of tissue inhibitor of metalloproteinase-1

American journal of reproductive immunology (New York, N.Y. : 1989) ·doi:10.1111/j.1600-0897.1998.tb00067.x

PROBLEM: Elevated levels of activated macrophages are associated with endometriosis, but their role in the etiology of the disease is uncertain. The current study was undertaken to examine whether activated macrophages could modulate perito…

1997

O-134 Gonadotrophin-releasing hormone agonist (GnRHa) therapy alters activity of plasminogen activators (PA), Matrix Metalloproteinases (MMP) and of their inhibitors (PAI and MMPI) in rat models for adhesion formation and endometriosis: Potential GnRHa-regulated mechanisms reducing adhesion formation

·doi:10.1016/s0015-0282(97)90766-4