Seiro Satohisa

No ORCID on file · 4 papers in corpus · active 2016-2020

Study types

  • other 3
  • article 1

Condition tags

  • endometriosis 3
other 2020
ASPP2 suppression promotes malignancy via LSR and YAP in human endometrial cancer
Histochemistry and cell biology ·doi:10.1007/s00418-020-01876-8

Apoptosis-stimulating p53 protein 2 (ASPP2) is an apoptosis inducer that acts via binding with p53 and epithelial polarity molecule PAR3. Lipolysis-stimulated lipoprotein receptor (LSR) is an important molecule at tricellular contacts, and …

other 2020
Possibility of Targeting Claudin-2 in Therapy for Human Endometrioid Endometrial Carcinoma
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1007/s43032-020-00230-6

Claudin-2 (CLDN-2) is a leaky-type tight junction protein, and its overexpression increases tumorigenesis of some types of cancer cells. In the present study, to examine the possibility of targeting CLDN-2 in the therapy for endometrioid en…

article 2016
ISP-37-2 A case report of multiple adenomyotic cysts derived from uterine serosa(Group 37 Women's Health 2,International Session Poster)
other 2016
The roles of tricellular tight junction protein lipolysis-stimulated lipoprotein receptor in malignancy of human endometrial cancer cells
Oncotarget ·doi:10.18632/oncotarget.8408

Lipolysis-stimulated lipoprotein receptor (LSR) has been identified as a novel molecular constituent of tricellular contacts that have a barrier function for the cellular sheet. LSR recruits tricellulin (TRIC), which is the first molecular …