Ann‐Muriel Steff

article 2004

Quantitative Assessment of Human Endometriotic Tissue Maintenance and Regression in a Noninvasive Mouse Model of Endometriosis

Molecular therapy : the journal of the American Society of Gene Therapy ·doi:10.1016/j.ymthe.2003.12.012

Endometriosis is a prevalent disease characterized by the estrogen-dependent ectopic growth of endometrial tissue. Most of the current medical therapies consist in inducing a hypoestrogenic state in patients, but these treatments are associ…

article 2004

Serum Concentrations of Insulin‐like Growth Factor‐1, Soluble Tumor Necrosis Factor Receptor‐1 and Angiogenin in Endometriosis Patients

American journal of reproductive immunology (New York, N.Y. : 1989) ·doi:10.1046/j.8755-8920.2003.00138.x

PROBLEM: Many soluble factors contributing to the pathophysiology of endometriosis are found at abnormal levels in patients suffering from the disease. We postulated that levels of these factors could also be altered in the serum of patient…

article 2003

An improved mouse model for endometriosis allows noninvasive assessment of lesion implantation and development

Fertility and sterility ·doi:10.1016/s0015-0282(03)00986-5

ObjectiveTo test whether fragments of human endometrium transduced with the green fluorescent protein (GFP) cDNA and transplanted into nude mice can be noninvasively visualized.DesignA murine experimental model for human endometriosis.Setti…

article 2003

Concentration of soluble intercellular adhesion molecule-1 in serum samples from patients with endometriosis collected during the luteal phase of the menstrual cycle

Human Reproduction ·doi:10.1093/humrep/deg514

BACKGROUND: Soluble intercellular adhesion molecule-1 (sICAM-1), released by endometriotic lesions, is involved in the regulation of cytotoxic processes. Altered levels of sICAM-1 in the circulation could parallel its deregulation in the pe…

article 2002

Establishment of an improved mouse model of endometriosis allowing the non-invasive assessment of implantation, progression and regression of human endometriotic lesions

·doi:10.1016/s0015-0282(01)03019-9