Ji B

No ORCID on file · 6 papers in corpus · active 2013-2025
2025
Unraveling the Microenvironment and the Pathogenic Axis of HIF-1α-Visfatin-Fibrosis in Autoimmune Pancreatitis Using a Single-Cell Atlas.
Advanced science (Weinheim, Baden-Wurttemberg, Germany) ·doi:10.1002/advs.202412282

Autoimmune pancreatitis (AIP) is identified as a severe chronic immune-related disorder in pancreas, including two subtypes. In this study, pancreatic lesions in patients diagnosed as either type 1 AIP or type 2 AIP are examined, and these …

2021
Pancreas-specific CHRM3 activation causes pancreatitis in mice.
JCI insight ·doi:10.1172/jci.insight.132585

Hyperstimulation of the cholecystokinin 1 receptor (CCK1R), a G protein-coupled receptor (GPCR), in pancreatic acinar cells is commonly used to induce pancreatitis in rodents. Human pancreatic acinar cells lack CCK1R but express cholinergic…

2021
Prediction of Multiple Organ Failure Complicated by Moderately Severe or Severe Acute Pancreatitis Based on Machine Learning: A Multicenter Cohort Study.
Mediators of inflammation ·doi:10.1155/2021/5525118

BackgroundMultiple organ failure (MOF) may lead to an increased mortality rate of moderately severe (MSAP) or severe acute pancreatitis (SAP). This study is aimed to use machine learning to predict the risk of MOF in the course of disease.M…

2020
Neuroendocrine tumors of the gallbladder.
Oncology letters ·doi:10.3892/ol.2020.11461

A gallbladder neuroendocrine neoplasm (GB-NEN) is a unique heterogeneous neoplasm, originating from neuroendocrine cells and able to secrete peptides as its neurotransmitter. Among all the neuroendocrine tumors (NETs), the GB-NEN prevalence…

2019
An Infertile Couple's Long and Expensive Path to Varicocele Repair.
Urology ·doi:10.1016/j.urology.2018.10.021

ObjectiveTo characterize the pathways to care that infertile couples requiring varicocele repair (VR) pursue prior to presenting to a male fertility clinic.MethodsAn IRB-approved single center retrospective review of patients undergoing VR …

2013
Activation of nuclear factor-κB in acinar cells increases the severity of pancreatitis in mice.
Gastroenterology ·doi:10.1053/j.gastro.2012.09.059

Background & aimsNuclear factor-κB (NF-κB) is activated during early stages of pancreatitis. This transcription factor regulates genes that control many cell activities, including inflammation and survival. There is evidence that activation…