Zheng-Chen Guo

No ORCID on file · 7 papers in corpus · active 2021

Study types

  • other 5
  • article 2

Condition tags

  • adenomyosis 7
  • dysmenorrhea 1
other 2021
Additional file 1 of Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
·doi:10.6084/m9.figshare.14600554.v1

Additional file 1: Supplementary Figure S1. Morphological features of adenomyotic eutopic and ectopic endometrial stromal cells. A.and B. The primary culture of ADS_Eu_ESC and ADS_Ec_ESC cells to the fourth day, respectively. C. The third g…

other 2021
Additional file 1 of Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
·doi:10.6084/m9.figshare.14600554

Additional file 1: Supplementary Figure S1. Morphological features of adenomyotic eutopic and ectopic endometrial stromal cells. A.and B. The primary culture of ADS_Eu_ESC and ADS_Ec_ESC cells to the fourth day, respectively. C. The third g…

other 2021
Elevated Circular RNA PVT1 Promotes Eutopic Endometrial Cell Proliferation and Invasion of Adenomyosis via miR-145/Talin1 Axis
BioMed research international ·doi:10.1155/2021/8868700

Several theories on the origin of adenomyosis (ADS) have been proposed, of which the most widely accepted is the fundamental pathogenic role of uterine eutopic endometrium. Emerging evidence suggests that circular RNAs participate in the mu…

article 2021
Talin1 Induces Epithelial-Mesenchymal Transition to Facilitate Endometrial Cell Migration and Invasion in Adenomyosis Under the Regulation of microRNA-145-5p
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1007/s43032-020-00444-8
other 2021
Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
·doi:10.6084/m9.figshare.c.5425468.v1

Abstract Adenomyosis (ADS) is an estrogen-dependent gynecological disease with unspecified etiopathogenesis. Local hyperestrogenism may serve a key role in contributing to the origin of ADS. Talin1 is mostly identified to be overexpressed a…

article 2021
Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
Reproductive biology and endocrinology : RB&E ·doi:10.1186/s12958-021-00756-7

Abstract Adenomyosis (ADS) is an estrogen-dependent gynecological disease with unspecified etiopathogenesis. Local hyperestrogenism may serve a key role in contributing to the origin of ADS. Talin1 is mostly identified to be overexpressed a…

other 2021
Upregulated Talin1 synergistically boosts β-estradiol-induced proliferation and pro-angiogenesis of eutopic and ectopic endometrial stromal cells in adenomyosis
·doi:10.6084/m9.figshare.c.5425468

Abstract Adenomyosis (ADS) is an estrogen-dependent gynecological disease with unspecified etiopathogenesis. Local hyperestrogenism may serve a key role in contributing to the origin of ADS. Talin1 is mostly identified to be overexpressed a…