Julie A. Birt

ORCID: 0000-0003-2105-2056 · 5 papers in corpus · active 2010-2013

Study types

  • article 2
  • dissertation 1
  • other 1
  • review 1

Condition tags

  • endometriosis 5
  • infertility 2
article 2013
Developmental exposure of fetal ovaries and fetal germ cells to endometriosis in an endometriosis model causes differential gene expression in the preimplantation embryos of the first-generation and second-generation embryos
Fertility and sterility ·doi:10.1016/j.fertnstert.2013.07.007
article 2013
Elevated Peritoneal Fluid TNF-α Incites Ovarian Early Growth Response Factor 1 Expression and Downstream Protease Mediators: A Correlation With Ovulatory Dysfunction in Endometriosis
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1177/1933719113477479
review 2012
Cellular and molecular basis for endometriosis-associated infertility
Cell and tissue research ·doi:10.1007/s00441-011-1309-0

Endometriosis is a gynecological disease characterized by the presence of endometrial glandular epithelial and stromal cells growing in the extra-uterine environment. The disease afflicts 10%-15% of menstruating women causing debilitating p…

dissertation 2010
Heritable, differential gene expression in pre-implantation embryos from rats with endometriosis and their daughters
·doi:10.32469/10355/10650

The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file.

other 2010
Neutralizing TIMP1 restores fecundity in a rat model of endometriosis and treating control rats with TIMP1 causes anomalies in ovarian function and embryo development
Biology of reproduction ·doi:10.1095/biolreprod.109.083287

Human and rat endometriotic lesions synthesize and secrete tissue inhibitor of metalloproteinase 1 (TIMP1). More TIMP1 localizes in the ovarian theca in an established rat model for endometriosis (Endo) when compared to surgical controls (S…