G Vinci

ORCID: 0000-0002-2968-3867 · 2 papers in corpus
article 2016
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1177/1933719116638188

The current study aimed to identify and validate an applicable immunohistochemistry panel including Ki-67, c-MYC, estrogen receptor-α (ER-α), and progesterone receptor isoforms A/B (PR-A/B) in correlation with clinicopathological parameters…

article 2010
Molecular Endocrinology ·doi:10.1210/me.2010-0160

Several lines of evidence indicate that endometriosis could be partially due to selective epigenetic deregulations. Promoter hypermethylation of some key genes, such as progesterone receptor and aromatase, has been associated with the silen…