Study types
- preprint 13
- article 1
Condition tags
- endometriosis 14
Frequent coauthors
- Xin Huang 14
- Esther Elishaev 14
- George C. Tseng 14
- Raluca A. Budiu 14
- Robert P. Edwards 14
- Swati Suryawanshi 14
- Anda Vlad 14
- Ted Lee 13
- Gina Mantia-Smaldone 13
- Suketu Mansuria 13
preprint
2023
Supplementary Materials and Methods
preprint
2023
Supplementary Materials and Methods
preprint
2023
Suppl Fig S2. Kras and Pten pathways in relationship to complement gene expression
preprint
2023
Suppl Fig S2. Kras and Pten pathways in relationship to complement gene expression
preprint
2023
Suppl Fig S3. Antibody-mediated, complement-induced cytotoxicity
preprint
2023
Suppl Fig S4. Complement and non-target siRNA
preprint
2023
Suppl Fig S4. Complement and non-target siRNA
preprint
2023
Suppl Table S1. Differentially expressed genes from different group comparisons
preprint
2023
Suppl Table S1. Differentially expressed genes from different group comparisons
preprint
2023
Suppl Table S2. Top five canonical pathways from different group comparisons, identified via Ingenuity Pathway Analyses
preprint
2023
Suppl Table S2. Top five canonical pathways from different group comparisons, identified via Ingenuity Pathway Analyses
preprint
2023
Suppl Table S3. Top five networks from different group comparisons, identified via Ingenuity Pathway Analyses
preprint
2023
Suppl Table S3. Top five networks from different group comparisons, identified via Ingenuity Pathway Analyses
article
2014
Abstract Endometriosis is a largely benign, chronic inflammatory disease that may predispose to some of the ovarian tumors histotypes, collectively called endometriosis-associated ovarian cancers (EAOC). Although it is widely accepted that …