Teasley HE

No ORCID on file · 5 papers in corpus
article 2020
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1007/s43032-019-00017-4

Accumulating research shows that ovarian cancer progression can be influenced by both gene mutations and endometriosis. However, the exact mechanism at hand is poorly understood. In the current study, we explored the expression of KRAS and …

preprint 2020
·doi:10.1101/2020.09.23.308528

Abstract Though endometriosis and infertility are clearly associated, the pathophysiological mechanism remains unclear. Previous work has linked endometrial ARID1A loss to endometriosis-related endometrial non-receptivity. Here, we show in …

other 2019
Biochemical and biophysical research communications ·doi:10.1016/j.bbrc.2019.02.034

The ovarian steroid hormones, estrogen (E2) and progesterone (P4), are essential regulators of uterine biology. The imbalance of these ovarian steroid hormones leads to uterine diseases such as endometrial cancer, endometriosis, and inferti…

2018
Biochemical and biophysical research communications ·doi:10.1016/j.bbrc.2017.12.154

The ovarian steroid hormones, estrogen (E2) and progesterone (P4), are essential regulators of uterine functions necessary for development, embryo implantation, and normal pregnancy. ARID1A plays an important role in steroid hormone signali…

2018
Asian-Australasian Journal of Animal Sciences ·doi:10.5713/ajas.18.0011

ObjectiveProgesterone receptor (PGR) and AT-rich interactive domain 1A (ARID1A) have important roles in the establishment and maintenance of pregnancy in the uterus. In present studies, we examined the expression of mitochondrial tumor supp…