Huang, Zhi-Xiong
No ORCID on file
· 10 papers in corpus
lab-animal
2025
Supplementary Material 1. Figure S1. The Construction and Identification of an EMs Mouse Model and Bioinformatic Analysis of EMs-Related Data. (A) Establishment of an endometriotic mouse model. (B) Tissue sections of ectopic lesions in EMs …
lab-animal
2025
Supplementary Material 1. Figure S1. The Construction and Identification of an EMs Mouse Model and Bioinformatic Analysis of EMs-Related Data. (A) Establishment of an endometriotic mouse model. (B) Tissue sections of ectopic lesions in EMs …
lab-animal
2025
Supplementary Material 2. Figure S2. The impact of FTO on the proliferation, migration, and invasion of Eu-ESCs and Ec-ESCs. (A-B) Cell proliferation of Eu-ESCs overexpressing FTO (A) and suppressing FTO (B) was measured by a CCK-8 assay. *…
lab-animal
2025
Supplementary Material 2. Figure S2. The impact of FTO on the proliferation, migration, and invasion of Eu-ESCs and Ec-ESCs. (A-B) Cell proliferation of Eu-ESCs overexpressing FTO (A) and suppressing FTO (B) was measured by a CCK-8 assay. *…
lab-animal
2025
Abstract Background Endometriosis (EMs) is a condition characterized by the growth of endometrial tissue outside the uterine cavity. Although this condition is benign, it has cancer-like features. N6-methyladenosine (m6A) is a common RNA mo…
lab-animal
2025
Abstract Background Endometriosis (EMs) is a condition characterized by the growth of endometrial tissue outside the uterine cavity. Although this condition is benign, it has cancer-like features. N6-methyladenosine (m6A) is a common RNA mo…
lab-animal
2017
Objective:Epithelial–mesenchymal transition (EMT) is essential for embryogenesis, fibrosis, and tumor metastasis. Aberrant EMT phenomenon has been reported in endometriotic tissues of patients with endometriosis (EM). In this study, we furt…
lab-animal
2017
Objective:Epithelial–mesenchymal transition (EMT) is essential for embryogenesis, fibrosis, and tumor metastasis. Aberrant EMT phenomenon has been reported in endometriotic tissues of patients with endometriosis (EM). In this study, we furt…
2017
Supplementary Material for Lipoxin A4 Suppresses Estrogen-Induced Epithelial–Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis by Rong-Feng Wu, Zhi-Xiong Huang, Jing Ran, Song-Juan Dai, Dian-Chao Lin, Tai-Wei Ng, Qing-Xi Che…
2017
Supplementary Material for Lipoxin A4 Suppresses Estrogen-Induced Epithelial–Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis by Rong-Feng Wu, Zhi-Xiong Huang, Jing Ran, Song-Juan Dai, Dian-Chao Lin, Tai-Wei Ng, Qing-Xi Che…