{"paper_id":"f8277a7b-27de-44c0-aec1-1b53258981aa","body_text":"References\nStanic AK, Kim M, Styer AK, Rueda BR. Dendritic cells attenuate the early establishment of endometriosis-like lesions in a murine model. Reprod Sci. 2014;21(10):1228–1236.\nNaqvi H, Ilagan Y, Krikun G, Taylor HS. Altered genomewide methylation in endometriosis. Reprod Sci. 2014;21(10):1237–1243.\nGovernini L, Carrarelli P, Rocha AL, et al. FOXL2 in human endometrium: hyperexpressed in endometriosis. Reprod Sci. 2014;21(10):1249–1255.\nCarr B, Dmowski WP, O’Brien C, et al. Elagolix, an oral GnRH antagonist versus subcutaneous depot medroxyprogesterone acetate for the treatment of endometriosis: effects on bone mineral density. Reprod Sci. 2014;21(11):1341–1351.\nAppleyard CB, Cruz ML, Hernández S, Thompson KJ, Bayona M, Flores I. Stress management affects outcomes in the pathophysiology of an endometriosis model [published online ahead of print July 11, 2014]. Reprod Sci. doi: 10.1177/1933719114542022.\nFanasiak J, Burns K, Slayden O, et al. Endometrial CXCL13 Expression is cycle regulated in humans and aberrantly expressed in humans and rhesus macaques with endometriosis [published online ahead of print July 16, 2014]. Reprod Sci. doi: 10.1177/1933719114542011.\nFranco-Murillo Y, Miranda-Rodríguez JA, Rendón-Huerta E, et al. Unremitting cell proliferation in the secretory phase of eutopic endometriosis: Involvement of pAkt and pGSK3β [published online ahead of print September 6, 2014]. Reprod Sci. doi: 10.1177/1933719114549843.\nRights and permissions\nAbout this article\nCite this article\nMaduro, M.R. In the Spotlight. Reprod. Sci. 22, 389–390 (2015). https://doi.org/10.1177/1933719115574252\nPublished:\nIssue date:\nDOI: https://doi.org/10.1177/1933719115574252","source_license":"CC0","license_restricted":false}