{"paper_id":"9881db4e-e8e3-456d-a55c-eaae71b00313","body_text":"References\nLiu Y, Sun L, Hou Z, Mao Y, Cui Y, Liu J. rhTNFR: Fc suppresses the development of endometriosis in a mouse model by downregulating cell proliferation and invasiveness. Reprod Sci. 2015. pii: 1933719115620495.\nLaganà AS, Sturlese E, Retto G, Sofo V, Triolo O. Interplay between misplaced Mu¨ llerian-Derived stem cells and peritoneal immune dysregulation in the pathogenesis of endometriosis. Obstet Gynecol Int. 2013;2013:527041.\nSturlese E, Salmeri FM, Retto G, et al. Dysregulation of the Fas/FasL system in mononuclear cells recovered from peritoneal fluid of women with endometriosis. J Reprod Immunol. 2011;92(1-2):74–81.\nRichter C, Messerschmidt S, Holeiter G, et al. The tumor necrosis factor receptor stalk regions define responsiveness to soluble versus membrane-bound ligand. Mol Cell Biol. 2012;32(13):2515–2529.\nSalmeri FM, Laganà AS, Sofo V, et al. Behavior of tumor necrosis factor-α and tumor necrosis factor receptor 1/tumor necrosis factor receptor 2 system in mononuclear cells recovered from peritoneal fluid of women with endometriosis at different stages. Reprod Sci. 2015;22(2):165–172.\nOthman ER, Hornung D, Hussein M, et al. Soluble tumor necrosis factor-alpha receptors in the serum of endometriosis patients. Eur J Obstet Gynecol Reprod Biol. 2016;20;200:1–5. doi:10.1016/j.ejogrb.2016.02.025.\nAuthor information\nAuthors and Affiliations\nCorresponding author\nRights and permissions\nAbout this article\nCite this article\nLaganà, A.S., Salmeri, F.M., Triolo, O. et al. Comments on “rhTNFR:Fc Suppresses the Development of Endometriosis in a Mouse Model by Downregulating Cell Proliferation and Invasiveness”. Reprod. Sci. 25, 983–984 (2018). https://doi.org/10.1177/1933719116653838\nPublished:\nVersion of record:\nIssue date:\nDOI: https://doi.org/10.1177/1933719116653838","source_license":"CC0","license_restricted":false}