{"paper_id":"68ef33b5-cb5b-4c19-a082-b072db10baf4","body_text":"Print ISSN: 1021-335X\nOnline ISSN: 1791-2431\nInternational Journal of Molecular Medicine is an international journal devoted to molecular mechanisms of human disease.\nInternational Journal of Oncology is an international journal devoted to oncology research and cancer treatment.\nCovers molecular medicine topics such as pharmacology, pathology, genetics, neuroscience, infectious diseases, molecular cardiology, and molecular surgery.\nOncology Reports is an international journal devoted to fundamental and applied research in Oncology.\nExperimental and Therapeutic Medicine is an international journal devoted to laboratory and clinical medicine.\nOncology Letters is an international journal devoted to Experimental and Clinical Oncology.\nExplores a wide range of biological and medical fields, including pharmacology, genetics, microbiology, neuroscience, and molecular cardiology.\nInternational journal addressing all aspects of oncology research, from tumorigenesis and oncogenes to chemotherapy and metastasis.\nMultidisciplinary open-access journal spanning biochemistry, genetics, neuroscience, environmental health, and synthetic biology.\nOpen-access journal combining biochemistry, pharmacology, immunology, and genetics to advance health through functional nutrition.\nPublishes open-access research on using epigenetics to advance understanding and treatment of human disease.\nAn International Open Access Journal Devoted to General Medicine.\nArticle\n- Authors:\n-\nPages: 37-40|Published online on: January 1, 1995https://doi.org/10.3892/or.2.1.37\n- Expand metrics +\nVirgin female SHN and SLN mice, a high and a low spontaneous mammary tumor strains, respectively, were given a single intravenous injection of streptozotocin (2 mg/10 g body weight), resulting in a significant rise of blood glucose levels and of mammary tumor incidence. The growth rate of mammary tumors was also accelerated by streptozotocin-induced diabetes in SHN mice, but not in SLN mice. The incidence and progression of uterine adenomyosis and adenocarcinoma were not affected by diabetes in SHN and SLN mice. These findings indicate that spontaneous mammary tumorigenesis in SHN and SLN mice is accelerated by streptozotocin-induced diabetes.\nCopy and paste a formatted citation\nSpandidos Publications style\nGOTO Y, NAGASAWA H and IGUCHI T: STREPTOZOTOCIN-INDUCED DIABETES ACCELERATES MAMMARY TUMORIGENESIS IN SHN AND SLN MICE. Oncol Rep 2: 37-40, 1995.\nAPA\nGOTO, Y., NAGASAWA, H., & IGUCHI, T. (1995). STREPTOZOTOCIN-INDUCED DIABETES ACCELERATES MAMMARY TUMORIGENESIS IN SHN AND SLN MICE. Oncology Reports, 2, 37-40. https://doi.org/10.3892/or.2.1.37\nMLA\nGOTO, Y., NAGASAWA, H., IGUCHI, T.\"STREPTOZOTOCIN-INDUCED DIABETES ACCELERATES MAMMARY TUMORIGENESIS IN SHN AND SLN MICE\". Oncology Reports 2.1 (1995): 37-40.\nChicago\nGOTO, Y., NAGASAWA, H., IGUCHI, T.\"STREPTOZOTOCIN-INDUCED DIABETES ACCELERATES MAMMARY TUMORIGENESIS IN SHN AND SLN MICE\". Oncology Reports 2, no. 1 (1995): 37-40. https://doi.org/10.3892/or.2.1.37","source_license":"public-domain-us","license_restricted":false}